The transversus linguae (transverse muscle of the tongue) is one of the four intrinsic tongue muscles. When this muscle involuntarily contracts or tightens, it causes a spasm—a sudden, often painful, contraction that can interfere with speaking, chewing, and swallowing. Wikipedia
Anatomy of the Transversus Linguae Muscle
Structure & Location
The transversus linguae is a thin sheet of fibers that runs horizontally from the midline septum of the tongue out toward its sides. It lies deep to the mucosa, interwoven with other intrinsic muscles. Wikipedia
Origin
Fibers originate from the median fibrous septum, a central connective tissue partition within the tongue. Wikipedia
Insertion
They pass laterally to insert into the submucosal fibrous tissue along the dorsum and sides of the tongue. Wikipedia
Blood Supply
The muscle receives arterial blood from branches of the lingual artery, specifically the deep lingual branches, ensuring oxygen delivery for rapid contraction and relaxation. NCBI
Nerve Supply
Motor innervation is via the hypoglossal nerve (cranial nerve XII), which controls all intrinsic and most extrinsic tongue muscles. Wikipedia
Functions
When the transversus linguae contracts, it:
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Narrows the tongue by pulling the sides inward.
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Elongates the tongue by increasing its front-to-back length.
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Shapes the tongue for precise articulation during speech.
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Stabilizes the tongue’s midline, aiding coordination of other muscles.
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Assists in bolus control during chewing.
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Guides the flow of saliva and food toward the pharynx. NCBI
Types of Transversus Linguae Muscle Spasms
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Acute Cramps – brief, intense, self‐limiting episodes often due to fatigue.
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Chronic Fasciculations – ongoing twitching without full contraction.
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Lingual Dystonia – sustained, involuntary contractions causing twisting postures (e.g., Meige syndrome). Cleveland Clinic
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Myoclonic Jerks – sudden, shock-like twitches of transversus fibers. WebMD
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Spasticity – increased muscle tone often secondary to neurological injury. Cleveland Clinic
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Idiopathic Spasms – spasms with no identifiable medical cause.
Causes
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Dehydration – electrolyte imbalance triggers muscle hyperexcitability.
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Hypocalcemia – low calcium levels cause neuromuscular irritability.
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Hypomagnesemia – magnesium deficiency leads to repeated contractions.
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Fatigue/Overuse – prolonged tongue use (e.g., singing) strains intrinsic fibers.
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Anxiety/Stress – increased muscle tension predisposes to spasms.
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Medication Side Effects – stimulants or diuretics may provoke cramps.
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Oral Trauma – injury to tongue tissue can irritate muscle fibers.
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Neurological Disorders – conditions like multiple sclerosis may cause spasticity.
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Dystonia Syndromes – Meige or oromandibular dystonia specifically affect tongue muscles. Cleveland Clinic
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Cerebrovascular Accident – stroke can lead to spasticity of tongue muscles.
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Upper Motor Neuron Lesion – results in hyperreflexive tongue contraction.
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Peripheral Nerve Injury – hypoglossal nerve damage can cause aberrant reinnervation.
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Metabolic Disturbances – thyroid or adrenal disorders affecting muscle function.
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Infections – tetanus or local abscess may irritate muscle tissue.
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Nutritional Deficiencies – lack of B vitamins or trace minerals.
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Acid Reflux – chronic irritation of oropharynx can trigger reflex spasms.
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Allergic Reactions – angioedema-like swelling may secondarily spasm.
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Dental Appliances – ill-fitting dentures can mechanically irritate the tongue.
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Alcohol/Drug Use – neurotoxic effects disrupt normal muscle control.
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Idiopathic – no identifiable underlying trigger. Cleveland Clinic
Symptoms
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Sharp, Stabbing Tongue Pain
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Tongue Tightness or Hardening
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Difficulty Speaking Clearly
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Intermittent Locking of the Tongue
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Involuntary Twisting or Bending
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Fasciculations (Twitching)
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Swallowing Difficulty (Dysphagia)
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Excessive Salivation
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Speech Distortion
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Burning Sensation
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Quivering of Tongue Tip
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Impaired Chewing
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Reflex Jaw Clenching
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Local Muscle Tenderness
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Fatigue After Speaking
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Fear of Speaking in Public (Secondary Anxiety)
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Reduced Taste Sensation
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Dry Mouth from Restricted Movement
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Pain Radiating to Jaw
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Nighttime Awakening by Spasms HealthlineCleveland Clinic
Diagnostic Tests
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Clinical Oral Examination
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Electromyography (EMG) – measures muscle electrical activity.
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Nerve Conduction Studies
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Serum Electrolyte Panel
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Calcium & Magnesium Levels
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Thyroid Function Tests
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MRI of Brainstem – rules out central lesions.
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CT Scan of Head and Neck
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Video Fluoroscopic Swallow Study
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Flexible Endoscopic Evaluation of Swallowing (FEES)
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Ultrasound of Tongue Musculature
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Laryngoscopy
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Cranial Nerve Function Assessment
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Blood Glucose Levels
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Autoimmune Panel
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Infectious Workup (e.g., tetanus antitoxin)
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Psychiatric Evaluation (if stress-related)
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Allergy Testing
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Dental/Oral Appliance Fit Assessment
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Speech-Language Pathology Assessment Cleveland Clinic
Non-Pharmacological Treatments
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Hydration & Electrolyte Repletion
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Warm Salt-Water Gargles
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Gentle Tongue Stretching Exercises
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Myofunctional Therapy (speech-language exercises)
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Massaging the Tongue Base
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Biofeedback Techniques
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Acupuncture
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Transcutaneous Electrical Nerve Stimulation (TENS)
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Yoga & Relaxation Practices Colgate
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Meditation & Deep Breathing
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Progressive Muscle Relaxation
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Chewing Sugar-Free Gum (sensory trick)
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Biting on a Tongue Depressor
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Cold Compress Application
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Speech Therapy
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Swallowing Therapy
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Avoidance of Irritants (spicy, acidic foods)
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Proper Oral Appliance Adjustment
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Neck & Jaw Stretching
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Tongue-Position Training
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Mirror-Guided Motor Re-training
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Mirror Therapy for Neuromuscular Control
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Cognitive Behavioral Therapy for Anxiety
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Weighted Tongue Balance Exercises
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Ultrasound-Guided Muscle Release
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Laser Acupuncture
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Manual Trigger-Point Release
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Dietary Adjustment (magnesium-rich foods)
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Avoid Caffeine & Alcohol
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Sleep Hygiene to Reduce Nocturnal Spasms Cleveland Clinic
Pharmacological Treatments
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Baclofen – GABA analog reducing spasticity.
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Clonazepam – benzodiazepine for myoclonic spasms. WebMD
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Diazepam – muscle relaxant for acute cramps.
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Trihexyphenidyl – anticholinergic for dystonia.
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Gabapentin – modulates calcium channels to reduce hyperexcitability.
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Carbamazepine – sodium-channel blocker for focal spasms.
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Tizanidine – alpha-2 agonist for muscle tone reduction.
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Botulinum Toxin Type A – focal chemodenervation in dystonia. Cleveland Clinic
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Triamcinolone Injections – reduces local inflammation.
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Clonidine – central alpha-agonist for spasm reduction.
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Levetiracetam – adjunct for myoclonic jerks.
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Diazepam Oral Rinse – topical muscle relaxant.
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Magnesium Supplements
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Calcium Gluconate IV – for acute hypocalcemic cramps.
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Dantrolene Sodium – direct muscle relaxant.
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Phenobarbital – barbiturate adjunct in refractory cases.
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Botulinum Toxin Type B – alternative serotype in resistant dystonia.
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Trihexyphenidyl Extended-Release – longer effect in chronic cases.
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Lithium – in certain refractory movement disorders.
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Pregabalin – for neuropathic component of spasms. WebMD
Surgical & Procedural Treatments
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Selective Peripheral Denervation – cutting overactive nerve branches.
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Microvascular Decompression – for nerve-entrapment spasticity.
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Partial Glossectomy – reducing muscle bulk in severe dystonia.
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Hypoglossal Nerve Neurectomy – interrupting aberrant signals.
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Deep Brain Stimulation (GPi Targeting) – for generalized dystonia.
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Radiofrequency Ablation of Motor Nerve Fibers
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Cryoablation of Intrinsic Muscle
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Intramucosal Botulinum Toxin Injection Cleveland Clinic
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Ultrasound-Guided Muscle Hydrodissection
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Myotomy of Transverse Muscle Fibers
Prevention Strategies
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Maintain Adequate Hydration
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Balance Electrolytes with Diet/Supplements
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Practice Regular Tongue Stretching
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Manage Stress Through Relaxation
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Avoid Excess Caffeine and Diuretics
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Ensure Proper Fitting of Oral Appliances
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Limit Overuse in Singing or Speaking
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Warm-Up Exercises Before Vocal Performance
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Engage in Myofunctional Therapy
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Regular Dental Check-Ups to Detect Irritants Cleveland Clinic
When to See a Doctor
See your healthcare provider if spasms:
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Happen frequently or last more than a few minutes.
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Cause severe pain, interference with eating, speaking, or breathing.
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Are accompanied by numbness, swelling, or neurological symptoms like weakness. HealthlineCleveland Clinic
Frequently Asked Questions
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What triggers tongue spasms?
Low electrolytes, stress, and overuse can all trigger spasms. -
Are tongue spasms dangerous?
Most are harmless but see a doctor if they injure the tongue or disrupt swallowing. -
Can hydration prevent spasms?
Yes—dehydration is a common cause, so drinking fluids helps. -
Is there a specific diet to reduce spasms?
A diet rich in magnesium, calcium, and B vitamins can lower risk. -
How long do spasms usually last?
Acute cramps last seconds to minutes; chronic dystonic spasms can last hours. -
Will speech therapy help?
Yes—exercises improve muscle control and reduce frequency. -
Can Botox be used for tongue spasms?
Yes—botulinum toxin injections relieve focal dystonia of the tongue. -
Are there self-care tips for immediate relief?
Gentle stretching, cold compresses, and salt-water gargles can ease spasms. -
When is surgery necessary?
Only in severe, treatment-resistant dystonia or nerve-entrapment cases. -
Do spasms indicate a neurological disease?
Sometimes—if accompanied by other muscle or neurological signs. -
Can anxiety cause tongue spasms?
Yes—stress and anxiety increase muscle tension. -
Are over-the-counter muscle relaxants effective?
Mild cases may respond, but prescription drugs are often needed. -
Is tongue spasm the same as lockjaw?
No—lockjaw (trismus) affects jaw muscles, not intrinsic tongue muscles. -
Can medications cause spasms?
Certain diuretics, stimulants, and neuro-active drugs are known triggers. -
How can I differentiate spasm from tremor?
Spasms are sustained contractions; tremors are rhythmic oscillations.
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Last Updated: April 24, 2025.