MOOKP is a special surgery for people who are blind from severe corneal and ocular surface disease and have very dry, scarred eyes. Surgeons take a small piece of the patient’s tooth and jawbone (usually a canine tooth) and carve it into a tiny plate (lamina). They insert a clear plastic optical cylinder (like a mini window/lens) through that plate. After a few months of letting this tooth-bone plate “get a blood supply” under the skin of the cheek, they implant the plate into the eye so light can enter through the cylinder and reach the retina. In many properly selected patients, this can restore useful sight when other surgeries cannot. EyeWiki
Doctors consider MOOKP for people who:
Are blind in both eyes from corneal and surface disease.
Have severe dryness and scarring where routine corneal transplants or other prostheses (like Boston KPro type 1) would likely fail.
Still have a working retina and optic nerve (the “wiring” at the back of the eye).
Surgeons remove scarred tissues from the eye and later cover the front of the eye with a buccal mucosa (cheek) graft so the surface becomes living, moist, and more durable than a dry scar.
Separately, a single-root tooth (usually a canine) with a surrounding sliver of jawbone is harvested. A polymethyl-methacrylate (PMMA) optical cylinder (the clear “window”) is embedded through a drilled channel in this tooth-bone plate (the osteoodonto-alveolar lamina).
That tooth-bone-plus-lens is first tucked under the skin near the eye to vascularize (grow a blood supply).
A few months later, it’s moved and fixed on the front of the eye, and the mucosal flap is closed over it so only the tiny tip of the lens protrudes. Light then passes through the cylinder to the retina. EyeWiki
How MOOKP works
Prepare the eye surface: The surgeon covers the damaged eye with a thin buccal (cheek) mucous membrane graft to create a healthy skin-like cover.
Select and harvest a tooth: A single-root tooth (usually an upper canine) with healthy bone is chosen after dental X-rays/CT. A maxillofacial surgeon removes a small block of tooth + surrounding bone.
Carve the lamina: The team drills a hole and fits a clear optical cylinder into the tooth-bone plate.
“Bank” the lamina under the skin: The plate is inserted under the skin of the cheek for weeks to months so it gets a blood supply and becomes biologically active.
Implant the lamina into the eye: The surgeon brings the plate out from the cheek, makes an opening in the eye’s front wall, inserts the optical cylinder, and positions the tooth-bone lamina securely. The cheek membrane remains as the eye’s protective surface, with the cylinder peeking through as the viewing port.
This two-stage plan, often called the Rome–Vienna protocol, is the standard method. KProClinical GateEyeWiki
Types
Original OOKP (Strampelli) vs. Modified OOKP (Falcinelli/MOOKP):
The “modified” method refined the steps, instruments, and aftercare to improve retention and vision over time. When people say MOOKP, they usually mean Falcinelli’s version. EyeWikiPubMedAutologous vs. Allogeneic lamina:
Autologous = using your own tooth and bone (preferred).
Allogeneic = using a relative’s tooth when you have no suitable tooth. This can work, but it needs immune-suppression and has higher long-term risks, so it’s reserved for special cases. Clinical Gate
Alternatives when no tooth is usable:
Some centers may consider a tibial bone substitute or other keratoprostheses (e.g., Boston KPro type 2 for severe dry eye), but patient selection is very different. MOOKP is often preferred for the driest, most scarred eyes when a good tooth is available. escrs.orgEyeWiki
In experienced hands and carefully chosen patients, MOOKP can give durable vision for many years, with high anatomical survival and useful functional results reported in long-term series. The classic Falcinelli paper reported good anatomical and visual outcomes in 181 cases with long follow-up. Later reviews confirm MOOKP’s long-term reliability in the right patients. PubMed+1
Glaucoma (damage from high eye pressure) remains a major threat in all keratoprosthesis eyes, so pressure control and lifelong follow-up are critical. EyeWiki
Causes
These are not causes of MOOKP itself, but reasons the eye becomes so dry and scarred that doctors consider MOOKP to restore sight:
Chemical burns (especially alkali like lime or cement): Cause deep scarring, blood vessel growth, and dryness that defeat standard grafts. Wikipedia
Thermal burns (heat injuries): Destroy the corneal surface and eyelids, leading to severe exposure and scarring. Wikipedia
Stevens–Johnson syndrome (SJS) / Toxic epidermal necrolysis (TEN): Severe immune reaction (often to drugs) causing lifelong dry, scarred ocular surface. enhancedvision.com
Ocular cicatricial pemphigoid (OCP / Mucous membrane pemphigoid): Autoimmune scarring disease of the conjunctiva; dryness and eyelid adhesions make standard transplants fail. EyeWiki
Trachoma scarring: Chronic infection causing lid and conjunctival scarring and surface failure. Wikipedia
Severe Sjögren’s-related dry eye: Extreme dryness with surface damage; in rare, end-stage cases, MOOKP may be considered. PMC
Chronic graft-versus-host disease (ocular GVHD): Post-transplant immune attack causing severe dryness and scarring. PMC
Multiple failed corneal grafts in a dry, vascularized cornea: Repeated transplant failure leads to prosthesis options. EyeWiki
Severe vitamin A deficiency (xerophthalmia): Can cause keratinization and ulcers; rare in developed settings but still seen globally. EyeWiki
Autoimmune overlap scarring disorders (e.g., linear IgA disease) affecting mucous membranes, including the eye. PMC
Radiation-induced ocular surface failure (after orbital/ocular radiotherapy). PMC
Severe atopic or herpetic keratopathy with surface breakdown and vascularization unresponsive to other care. EyeWiki
Severe thermal lid deformities causing exposure keratopathy and keratinization. EyeWiki
Chemical warfare/industrial accidents with devastating ocular surface loss. EyeWiki
Limbal stem cell deficiency (various causes) with total surface failure and dryness. EyeWiki
Severe ocular surface tumors treated with aggressive therapy leading to scarring and dryness. EyeWiki
Severe allergic cicatrizing conjunctivitis (rare, end-stage). EyeWiki
Congenital aniridia with end-stage LSCD (rare cases where dryness/scarring dominate and other grafts fail). EyeWiki
Severe exposure keratopathy from facial nerve palsy with scarring when other reconstructions fail. EyeWiki
Long-standing, dense corneal vascularization and keratinization of any cause where standard grafts will not survive. EyeWiki
(These are examples reported across keratoprosthesis literature; the exact indication depends on individual examination and center experience.)
Symptoms
Severe blurry vision or blindness: The world looks foggy, dim, or black because light cannot pass through the scarred cornea.
Pain or burning: The surface is damaged and dry, so it hurts.
Extreme dryness/grittiness: Feels like sand in the eyes all the time.
Redness and irritation: The surface is inflamed and unstable.
Light sensitivity (photophobia): Bright light hurts and vision washes out.
Mucus discharge or crusting: The eye tries to protect itself with sticky secretions.
Recurrent erosions or ulcers: The top layer keeps breaking down.
Eyelids sticking to the eye (especially on waking): Due to dryness and scarring.
Frequent infections: A damaged surface invites germs.
Lashes rubbing the eye (trichiasis): Scarred lids point lashes inward, scratching the cornea.
Lid scarring and adhesions (symblepharon): The inner eyelid sticks to the eye surface.
Decreased tear meniscus (low tear lake): You can even see the lack of tears.
Poor blinking comfort: Each blink scrapes a dry, rough surface.
Milky or white pupil area: The cornea is opaque or keratinized.
Reduced night vision: Scattering and opacity make dim light vision worse.
(Symptoms come from the underlying disease—the operation is chosen when these problems are so severe that standard treatments fail.)
Diagnostic Tests
A) Physical exam (what the doctor looks for at the chair)
Visual acuity check (even counting fingers): Measures how much you can see now and sets a baseline to compare after surgery.
Pupil reactions and RAPD test: Checks optic nerve function; if the nerve is badly damaged, vision cannot improve much even with a clear window.
Slit-lamp exam of lids/conjunctiva/cornea: Finds scarring, blood vessels, keratinization, lashes rubbing the eye, and ulcers.
Eyelid and lash evaluation: Looks for entropion, trichiasis, and lid shortening that will need correction.
Cheek (buccal) mucosa exam: Surgeons need healthy cheek lining to graft over the eye in Stage 1.
Dental/oral exam: A maxillofacial surgeon checks which tooth is healthy and strong enough to harvest (usually an upper canine). PMC
B) Manual tests (simple office procedures)
Schirmer’s test: A small paper strip measures tear production; very low values confirm severe dryness.
Tear breakup time (TBUT): Dye shows how quickly tears break apart—fast breakup means poor tear film.
Surface staining (fluorescein / lissamine green): Highlights raw spots and unstable epithelium.
Corneal sensation (Cochet-Bonnet esthesiometer): Tells if corneal nerves still work.
Lacrimal irrigation (syringing): Checks if tear drainage passages are blocked.
IOP by palpation or trans-palpebral tonometry: With an opaque cornea, doctors may feel pressure or use non-contact methods because standard devices can be unreliable on scarred surfaces.
C) Lab & pathology (to prove the diagnosis and plan care)
Conjunctival biopsy with direct immunofluorescence (DIF): The gold-standard to confirm OCP/MMP; it looks for immune proteins along the basement membrane. Note: it’s not positive in everyone, so the team chooses the biopsy site carefully and may repeat it. EyeWikiPMC
Autoimmune blood tests (e.g., anti-BP180, anti-laminin-332, ANA): Support the diagnosis and guide systemic treatment; results can be negative even when OCP is present. PMC
General pre-anesthesia labs (CBC, glucose, renal, liver tests): Make sure you’re fit for long surgeries and postoperative medicines.
Vitamin A level (if malnutrition suspected): Confirms treatable deficiency in xerophthalmia.
Cultures if infection is active: Helps choose antibiotics before any grafting or implantation.
D) Electrodiagnostic (to check the “wiring” to the brain)
Electroretinography (ERG): Measures retina health. A better ERG usually predicts better postoperative vision after keratoprosthesis. PubMed
Visual Evoked Potential (VEP): Checks the pathway from eye to brain. A useful VEP suggests the optic nerve can carry signals after surgery. PubMed
Electro-oculography (EOG) or related tests (as needed): Additional tools to assess retinal pigment epithelium if the team needs more information.
E) Imaging (to see behind the opaque cornea and choose the tooth)
B-scan ocular ultrasound: Shows the retina, macula, and vitreous when the cornea is opaque; helps predict visual potential and find problems like retinal detachment. e-mjm.org
Ultrasound biomicroscopy (UBM): Looks at the front of the eye—angle, residual iris, ciliary body—to plan surgery.
Optical coherence tomography (OCT) of the retina (if any window exists): Gives a high-detail picture of the macula to predict likely vision.
Dental panoramic X-ray (OPG): A “big picture” X-ray to select the best tooth and study the root and surrounding bone. KPro
Cone-beam CT of jaw/teeth: 3-D scan that precisely measures root size, direction, and bone quality, improving tooth selection. PMC
Special CT of the implanted lamina (after surgery): Some centers use electron-beam tomography or similar to monitor bone/dentine thickness and catch early resorption that could threaten the device. PMC
Endoscopic viewing (during surgery) of the back of the eye: Surgeons sometimes use intraoperative endoscopy to inspect the retina when the cornea is opaque, making sure the back of the eye is healthy enough to justify the implant. ResearchGate
Non-pharmacological treatments and supports
Each item below explains what it is, why it’s used, and the core mechanism in plain English.
Strict oral hygiene
What/Why: Gentle toothbrushing (opposite the harvest site at first), flossing, and antiseptic mouth care keep the oral graft and harvest wounds clean.
Mechanism: Lowers bacterial load, reducing infection risk at the buccal mucosa and tooth sites that are critical for implant stability. EyeWikiSmoking cessation
Why: Smoking impairs blood supply and wound healing in oral and ocular tissues; it increases infection and resorption risk.
Mechanism: Better oxygenation and microvascular perfusion improve graft take and lamina health. EyeWikiProtective eye shield (especially during sleep)
Why: Prevents accidental rubbing/pressure on the lens tip and mucosa.
Mechanism: Physical barrier reduces mechanical trauma while tissues integrate. EyeWikiHead elevation & supine rest early post-op
Why: Reduces pressure and helps the air bubble stabilize the anterior chamber right after Stage 2.
Mechanism: Improves fluid dynamics and wound stability. EyeWikiHumidified environment
Why: The mucosal surface likes moisture; dry air can crack or irritate it.
Mechanism: Humidity preserves epithelial hydration and comfort (adjunct to mucosal graft benefits). EyeWikiUV-blocking protective eyewear outdoors
Why: Mucosa and exposed cylinder tip can be light-sensitive; reducing glare and UV helps comfort and tissue health.
Mechanism: Lowers phototoxic stress and protects from windborne debris. (General cornea/ocular surface practice.)Hands-off discipline
Why: No eye rubbing.
Mechanism: Avoids micro-shear on the mucosa and shifting of the optical cylinder (mechanical protection best practice).Clean water & shower habits
Why: Unsterile water (pools, hot tubs) carries microbes.
Mechanism: Reduces pathogen exposure to the mucosa and cylinder.Scheduled long-term follow-up (lifelong)
Why: Glaucoma and lamina changes can develop years later.
Mechanism: Regular IOP strategy and periodic CT or targeted imaging detect lamina resorption early; timely revision prevents failure. EyeWikiDental/maxillofacial checks
Why: Harvest site and oral health affect lamina durability.
Mechanism: Treats caries/gingivitis promptly; maintains oral biome balance that supports graft health. EyeWikiNutritional optimization
Why: Protein, vitamins C/A, zinc support wound healing and mucosal health.
Mechanism: Supplies amino acids and cofactors for collagen and epithelial repair. (General surgical nutrition best practice.)Glycemic control (in diabetes)
Why: Poor glucose control delays wound healing and increases infection risk.
Mechanism: Reduces leukocyte dysfunction and microvascular damage.Psychological support
Why: MOOKP is life-changing and cosmetically different; anxiety/depression can reduce adherence.
Mechanism: Counseling improves coping and treatment adherence, which correlates with outcomes. EyeWikiCaregiver training
Why: Family often helps with drop schedules, shield handling, and hygiene.
Mechanism: Shared skill reduces errors and missed red flags (e.g., sudden blur, pain).Jaw/cheek care instructions
Why: Oral harvest can leave tightness or numbness.
Mechanism: Gentle range-of-motion and soft diet early reduce strain and support healing. EyeWikiAvoid contact sports / high-impact activities (initial months)
Why: Direct blows risk cylinder displacement.
Mechanism: Minimizes traumatic forces while tissues integrate.Home safety adjustments
Why: Depth perception can be different; early bump hazards are common.
Mechanism: Good lighting and clear walkways reduce falls and eye bumps.Medication adherence tools
Why: Complex regimens (antibiotics, steroids, IOP control).
Mechanism: Timers/apps increase adherence; fewer complications.Prompt treatment of eyelid/ocular surface infections
Why: Blepharitis or mucosal infection can threaten the implant.
Mechanism: Early hygiene and medical therapy limit bacterial bio-burden near the cylinder.Center-of-excellence pathway
Why: MOOKP outcomes improve in specialized teams that follow the Rome–Vienna protocol.
Mechanism: Standardized selection, technique, and follow-up lower failure. EyeWiki
Commonly used medicines
Important: Protocols vary by center. Dosing below reflects widely used surgical/ophthalmic practice or guideline-based peri-operative prophylaxis; your own team’s plan may differ for safety reasons.
Cefazolin (IV) – First-line peri-operative antibiotic prophylaxis
Class: First-generation cephalosporin.
Typical dose/time: 2 g IV within 60 minutes before incision; re-dose during long cases as per protocol.
Purpose: Reduce surgical-site infections.
Mechanism: Inhibits bacterial cell wall synthesis.
Key side effects: Allergy, GI upset. ASHPStanford MedicineClindamycin (IV) – Alternative if β-lactam allergy
Typical dose/time: 900 mg IV within 60 minutes pre-incision; re-dose per protocol.
Purpose/Mechanism/SE: Gram-positive/anaerobe coverage; protein synthesis inhibitor; diarrhea/C. difficile risk. ASHPStanford MedicinePrednisone (oral) – Systemic steroid after stages
Typical regimen: 0.5–1 mg/kg/day, short course then taper as directed.
Purpose: Control post-op inflammation and immune reactivity around the graft/cylinder.
Mechanism: Broad anti-inflammatory gene modulation.
SE: Hyperglycemia, mood change, infection risk, gastric irritation. EyeWikiPrednisolone acetate 1% (eye drops)
Typical use: 4–8×/day then taper (surgeon-directed).
Purpose: Local inflammation control.
Mechanism: Topical corticosteroid.
SE: IOP rise (monitor), cataract (less relevant post-aphakia). (Standard cornea practice.)Moxifloxacin 0.5% (eye drops) or equivalent broad-spectrum topical antibiotic
Typical use: 4×/day for 1–2 weeks around stages.
Purpose: Lower ocular surface bacterial load.
Mechanism: DNA gyrase/topoisomerase inhibition.
SE: Local irritation; rare allergy. EyeWikiAcetazolamide (oral)
Typical dose: 250 mg 2–4×/day short-term as needed.
Purpose: IOP reduction when pressure rises and topical drops are less effective under mucosa.
Mechanism: Carbonic anhydrase inhibition → less aqueous humor production.
SE: Tingling, fatigue, kidney stones; avoid if sulfa allergy. EyeWikiChlorhexidine 0.2% (mouthwash)
Typical use: 2–3×/day after oral procedures.
Purpose: Oral antisepsis to protect buccal graft and harvest sites.
Mechanism: Disrupts bacterial membranes.
SE: Taste change, rare staining. EyeWikiOmeprazole (oral) during systemic steroids
Typical dose: 20–40 mg daily while on higher-dose steroids.
Purpose: Gastric protection.
Mechanism: Proton pump inhibition.
SE: Headache, rare hypomagnesemia (long use).Timolol 0.5% (eye drops) if surgeon allows
Typical use: 1–2×/day; note: absorption through mucosa is different; many centers lean on oral IOP control and glaucoma surgery if needed.
Purpose/Mechanism: β-blocker lowers aqueous production.
SE: Bradycardia, bronchospasm (systemic absorption possible). EyeWikiAnalgesics (e.g., acetaminophen)
Typical dose: 500–1000 mg every 6–8 h (max 3–4 g/day, adjust for liver disease).
Purpose: Pain control after oral/ocular surgery.
Mechanism: Central prostaglandin effects.
SE: Hepatotoxicity if overdosed.
Why not list “dental prophylaxis amoxicillin” here? MOOKP involves oral surgery and centers often follow broader surgical prophylaxis guidance rather than routine dental endocarditis prophylaxis rules; your team will tailor this (β-lactam allergy alternatives include clindamycin). ASHPAAPD
Dietary / molecular and supportive supplements
Always clear supplements with your team; some interact with anesthetics, anticoagulants, or steroids.
Protein (1.2–1.5 g/kg/day) – building blocks for mucosa and wound collagen; speeds repair.
Vitamin C (500–1000 mg/day) – cofactor for collagen cross-linking; antioxidant for wound healing.
Vitamin A (3,000–5,000 IU/day) – supports mucosal epithelium health (avoid high doses in pregnancy).
Zinc (15–30 mg/day elemental) – enzyme cofactor for tissue repair; deficiency impairs healing.
Omega-3 fatty acids (1–2 g/day EPA+DHA) – dampen surface inflammation; may improve comfort.
Hydration goal (urine pale yellow) – supports tear film and mucosal moisture.
Arginine (6–9 g/day split) – conditionally essential in healing; supports collagen deposition.
Probiotics (per label) – may lower antibiotic-associated GI upset during systemic courses.
Calcium + Vitamin D – protects bone while on longer steroid courses; supports oral/jaw health.
B-complex (esp. B12/folate) – supports epithelial turnover and nerve health.
Selenium (100–200 mcg/day) – antioxidant cofactor; do not exceed upper limits.
Copper (1–2 mg/day) – cofactor for cross-linking enzymes in collagen; balance with zinc.
Collagen peptides (5–10 g/day) – supply glycine/proline for connective tissue (adjunctive).
L-carnitine (500–1000 mg/day) – may reduce fatigue during recovery; data modest.
Vitamin K2 (as advised) – balances bone turnover (if prolonged steroids used).
(Evidence for wound-healing nutrition is stronger for protein, vitamin C, zinc; others are supportive and should be individualized.)
Regenerative / biologic” adjuncts sometimes discussed
These are adjuncts or alternatives used before/around MOOKP in severe ocular-surface disease—not replacements for MOOKP when it’s clearly indicated. Use only under specialist care.
Autologous serum tears (20–50%) – patient’s own serum diluted as drops; provides growth factors and vitamin A to support epithelial health in severe dryness/neurotrophic states.
Platelet-rich plasma (PRP) eye drops – concentrated platelets release healing factors; used off-label for persistent epithelial problems.
Umbilical cord/placental serum drops (investigational/availability varies) – rich in epitheliotrophic factors.
Recombinant human nerve growth factor (cenegermin) – approved for neurotrophic keratitis; can improve corneal nerve health in select cases beforehand.
Topical cyclosporine/tacrolimus – immunomodulators that can calm surface inflammation, sometimes as a pre-MOOKP conditioning step in less cicatrized cases.
Limbal stem-cell–based procedures (CLET/SLET/COMET) – true surgical alternatives tried before MOOKP when there’s residual tear function; MOOKP is usually chosen after these options fail or are unsuitable. (General consensus across reviews.) PubMedScienceDirect
Surgeries you may hear about
MOOKP Stage 1/Intermediary/Stage 2 (the core operation) – described above; performed in specialized centers using Rome–Vienna protocol refinements for consistent results. EyeWiki
Mucosal revision (trimming or re-grafting) – done if the mucosa overgrows the cylinder, ulcerates, or threatens extrusion; this is among the most common re-operations after MOOKP. PubMed
Glaucoma surgery (valve implantation or cyclophotocoagulation) – required if pressure stays high despite medicine; crucial to protect the optic nerve long-term. EyeWiki
Vitreoretinal surgery – treats complications like retinal detachment or vitreous hemorrhage that can occur in some eyes with severe disease and after complex intraocular steps. PubMed
Alternative keratoprostheses or adjunctive salivary-gland transplant – Boston KPro is used in different indications; submandibular salivary gland (SMG) transplantation is another specialist option for extreme dry eye in selected cases (often separate from MOOKP). NaturePMC
Ways to prevent getting to a MOOKP-level problem
Immediate, expert care for chemical/thermal eye injuries; rinse copiously and seek emergency ophthalmology.
Early referral for severe SJS/TEN or ocular mucous membrane pemphigoid to control disease activity and scarring.
Avoid high-risk chemicals at work/home; use goggles and safety training.
Treat severe dry eye aggressively before scarring sets in; don’t ignore non-healing erosions.
Stop smoking to protect oral and ocular tissues.
Maintain dental health; poor oral status complicates MOOKP planning.
Control systemic diseases (diabetes, autoimmune disease) that impair healing.
Keep all follow-ups; long-term surveillance catches glaucoma and lamina changes early.
Practice infection hygiene (handwashing, safe water) around the eye.
Wear eye protection in dusty/windy work and during activities that risk facial trauma.
When to see a doctor urgently
Sudden drop in vision, a new dark curtain, or bursts of floaters/flashes (possible retinal issue).
Severe, deep eye pain or headache with nausea (possible high IOP).
Increasing redness, discharge, fever, or foul taste/odor in the mouth (possible infection).
Visible shift or wobble of the lens tip, or mucosa that looks whitish, raw, or broken down.
Any new trauma to the eye or face. (General post-op warning set; see your team’s instructions.) EyeWiki
What to eat (and what to avoid) during early recovery
Eat:
Soft, high-protein foods (eggs, yogurt, dal, fish, tofu) to protect oral harvest sites and supply building blocks.
Vitamin-C-rich options (citrus, guava, peppers) and zinc sources (lentils, seeds) for wound healing.
Plenty of fluids; aim for pale-yellow urine.
Avoid (initially):
Hard/chewy foods (nuts, tough meats) on the harvest side; gradually reintroduce per surgeon’s advice.
Very hot, spicy, or acidic foods that sting oral wounds.
Alcohol and tobacco, which slow healing and raise infection risk. (General surgical nutrition practice.)
FAQs
1) Is MOOKP the same as “tooth-in-eye” surgery?
Yes. That nickname describes the tooth-bone plate that holds the tiny lens. moorfields.nhs.uk
2) Why use my own tooth and bone?
Your tissues integrate and resist rejection; the bone/dentin hold the lens mechanically and biologically. EyeWiki
3) How long can it last?
Published series show high anatomic survival beyond 10–20 years in many patients when follow-up is strict. PubMed+1
4) Will I see “normally”?
Many regain functional vision (reading large print, mobility, faces), some reach driving-level vision; others remain limited depending on the retina/optic nerve and complications. PubMed
5) What is the biggest long-term risk?
Glaucoma (and how we monitor/manage it) and lamina resorption. EyeWiki
6) Can I wear glasses or contacts after MOOKP?
Glasses for fine-tuning focus or glare are common; soft contacts are generally not used over the cylinder.
7) Can I shower and swim?
Shower with care (eyes closed, avoid direct spray). No swimming/hot tubs until your team clears it. (Infection risk.)
8) Will the eye look normal?
The mucosa looks pink and moist; a small clear cylinder is visible—cosmetically different but stable.
9) Do I still need eye drops?
Yes—antibiotics and steroids initially, then tailored maintenance and IOP meds as needed. EyeWiki
10) What if I don’t have a suitable tooth?
Alternatives (other keratoprostheses) may be discussed; some centers explore substitute bone materials, but classic MOOKP uses your own tooth. PubMed
11) Why is this done only in a few hospitals?
It’s highly specialized and needs a trained multidisciplinary team plus long-term follow-up infrastructure. bmjophth.bmj.com
12) Will I need more surgery later?
Often yes—mucosal trimming or other revisions are common maintenance procedures. PubMed
13) How is pain handled?
Usually with oral analgesics; oral sites can be sore for a short while.
14) Do I need antibiotics for dental procedures later?
Your surgical team will guide this; peri-operative surgical prophylaxis is standard for MOOKP stages; future dental work follows standard dental-prophylaxis principles only if indicated for your medical profile. ASHPAAPD
15) What happens if the lamina weakens?
Your team monitors with exams and CT; early detection allows revision before failure. EyeWiki
Disclaimer: Each person’s journey is unique, treatment plan, life style, food habit, hormonal condition, immune system, chronic disease condition, geological location, weather and previous medical history is also unique. So always seek the best advice from a qualified medical professional or health care provider before trying any treatments to ensure to find out the best plan for you. This guide is for general information and educational purposes only. Regular check-ups and awareness can help to manage and prevent complications associated with these diseases conditions. If you or someone are suffering from this disease condition bookmark this website or share with someone who might find it useful! Boost your knowledge and stay ahead in your health journey. We always try to ensure that the content is regularly updated to reflect the latest medical research and treatment options. Thank you for giving your valuable time to read the article.
The article is written by Team RxHarun and reviewed by the Rx Editorial Board Members
Last Updated: August 13, 2025.
