Alice in Wonderland Syndrome (AIWS) is a rare neurological condition that makes a person’s perceptions of their own body or the world around them feel distorted. People with AIWS might see objects as much larger or smaller than they really are, or feel that parts of their body are changing size or shape. These distortions can also affect how time seems to pass or how sounds and touches feel. Although the exact cause remains unclear, AIWS episodes are usually brief—lasting from a few minutes up to an hour—and most people fully recognize that what they’re experiencing isn’t real. Health experts believe these symptoms arise from unusual patterns of electrical activity or blood flow in brain areas that process sensory information, especially the temporal-parietal-occipital junction where visual and body-sense pathways meet Wikipedia.
Alice in Wonderland Syndrome (AIWS), also known as Todd’s syndrome or dysmetropsia, is a rare neurological disorder characterized by transient distortions of perception. Individuals may experience micropsia (objects appear smaller), macropsia (objects appear larger), pelopsia (objects seem closer), teleopsia (objects seem farther), metamorphopsia (altered shapes), or tachysensia (time feels sped up or slowed) Wikipedia. Episodes are typically brief—lasting minutes to an hour—and can affect vision, touch, hearing, body image, and time perception.
AIWS most often arises in children and adolescents, though adults can be affected. The exact cause remains unclear, but AIWS is frequently linked to migraines, epilepsy, viral infections (particularly Epstein–Barr virus), head trauma, or psychoactive substances Wikipedia. MRI and EEG are usually normal, so diagnosis rests on clinical history and exclusion of other conditions. While distressing, AIWS itself is generally benign and self-limiting; management focuses on the underlying trigger Cleveland ClinicPMC.
Types
AIWS symptoms are often grouped into three main types based on the nature of the distortions:
Type A: Self-Perception Disturbances
In Type A, individuals experience changes in how their own body feels or looks. They might feel that part of their body is growing too large (macrosomatognosia) or shrinking too small (microsomatognosia), or even feel split into two separate halves (somatopsychic duality). These body-focused distortions sometimes come with feelings of being detached from one’s thoughts or surroundings (depersonalization or derealization) Wikipedia.
Type B: Visual Distortions
Type B involves changes in seeing the external world. Common experiences include micropsia (objects appear smaller), macropsia (objects appear larger), pelopsia (objects look closer), and teleopsia (objects look farther away). These “Lilliputian hallucinations” directly mirror the surreal events in Lewis Carroll’s novel which gave the syndrome its name Wikipedia.
Type C: Combined Disturbances
Type C is when someone has both Type A and Type B symptoms, experiencing simultaneous body-image and visual distortions. This combined form can be more unsettling because it affects multiple senses at once, blending strange body sensations with warped visual perceptions Wikipedia.
Causes
Epstein–Barr Virus Infection
AIWS often shows up during mononucleosis caused by the Epstein–Barr virus. The infection can inflame brain regions that process sensory input, leading to temporary perceptual distortions Wikipedia.Varicella-Zoster Virus Infection
Chickenpox or shingles can trigger AIWS when the varicella-zoster virus affects the brain’s sensory areas, causing episodes of warped perception Wikipedia.Influenza Infection
Flu viruses, especially severe cases, sometimes lead to brain inflammation. This can disrupt normal sensory processing and bring on AIWS episodes Wikipedia.Zika Virus Infection
Zika has been linked to neurological complications; in some rare pediatric cases, it can cause AIWS through its effects on brain tissue Wikipedia.Coxsackievirus Infection
Hand-foot-mouth disease or viral meningitis from Coxsackievirus can inflame sensory pathways, leading to brief AIWS symptoms Wikipedia.Malaria (Plasmodium falciparum)
Severe malaria can affect brain perfusion and electrical activity, and in rare cases, patients report AIWS-like distortions Wikipedia.Mycoplasma pneumoniae Infection
This atypical pneumonia bacterium can lead to neurological complications, including rare AIWS episodes, through immune-mediated inflammation Wikipedia.Streptococcus pyogenes Infection
Scarlet fever or strep throat complicated by streptococcal invasion of the brain can sometimes trigger AIWS symptoms Wikipedia.Migraines
Many adults with AIWS experience it during migraine attacks. Changes in blood flow or waves of electrical depolarization in the visual cortex can distort perception Wikipedia.Head Trauma
A blow to the head can injure areas that integrate visual or body-sense information, resulting in AIWS episodes as those areas heal Wikipedia.Brain Lesions (Tumors or Strokes)
Space-occupying lesions or stroke in the occipital, parietal, or temporal lobes can distort sensory inputs and cause AIWS symptoms Wikipedia.Epilepsy
Electrical storms in temporal lobe epilepsy can spread to sensory areas and produce transient perceptual distortions characteristic of AIWS Wikipedia.Psychoactive Drug Use
Substances like LSD, magic mushrooms, or high-dose marijuana can produce temporary AIWS-like hallucinations by altering neurotransmitter activity Wikipedia.Genetic Predisposition
Although not fully understood, some families show clusters of AIWS symptoms, suggesting inherited vulnerability in sensory processing pathways Wikipedia.Topiramate Toxicity
In rare cases, the migraine-preventing drug topiramate has been linked to AIWS episodes, likely through its effects on brain electrical activity Wikipedia.
Symptoms
Micropsia
You see objects as much smaller than they really are, as if everything shrank around you Wikipedia.Macropsia
Objects look much larger, making people appear giants in your visual world Wikipedia.Pelopsia
Things feel closer than they truly are, warping distance perception Wikipedia.Teleopsia
Objects seem farther away than they actually are, as if seen through a long tunnel Wikipedia.Metamorphopsia
Shapes of objects bend or twist, turning straight lines into waves or curves Wikipedia.Tachysensia
Your sense of time speeds up or slows down dramatically, making minutes feel like hours or vice versa Wikipedia.Depersonalization
You feel detached from your own body or thoughts, like watching yourself from outside Wikipedia.Derealization
The world seems dreamlike or unreal, as if you’re in a distorted movie Wikipedia.Nausea
Distortions can trigger queasy feelings, especially during migraine-related AIWS Wikipedia.Dizziness
Spinning or lightheaded sensations often accompany visual and body-image distortions Wikipedia.
Diagnostic Tests
Physical Examination
General Physical Exam
A doctor checks vital signs (blood pressure, heart rate, temperature) and overall health to rule out fever or systemic illness Wikipedia.Neurological Exam
This evaluates muscle strength, coordination, reflexes, and sensory responses to detect other neurological issues Wikipedia.Vision Exam (Snellen Chart)
Measuring how clearly you can read letters at a distance helps rule out primary eye problems Wikipedia.Pupillary Reflex Testing
Shining a light in the eyes checks pupil constriction and dilation, indicating healthy optic and oculomotor pathways Wikipedia.Mental Status Exam
Simple questions assess memory, attention, and orientation to ensure cognitive functions are intact Wikipedia.
Manual Neurological Tests
Finger-to-Nose Test
You touch your nose with each finger in turn—tests coordination between vision and movement Wikipedia.Heel-to-Shin Test
Sliding your heel down the opposite shin checks lower-limb coordination and balance Wikipedia.Two-Point Discrimination
Using a special tool, the examiner touches your skin at two points to see how well you can sense separate touches Wikipedia.
Lab and Pathological Tests
Complete Blood Count (CBC)
Measures red and white blood cells to detect infection, inflammation, or anemia Wikipedia.Metabolic Panel
Checks electrolytes, kidney, and liver function to find metabolic causes of altered brain function Wikipedia.Viral Serology (EBV Antibodies)
Blood tests detect recent Epstein–Barr infection, a common cause of pediatric AIWS Wikipedia.Cerebrospinal Fluid Analysis
A lumbar puncture examines CSF for signs of infection or inflammation in the brain and spinal cord Cleveland Clinic.
Electrodiagnostic Tests
Electroencephalogram (EEG)
Records brain electrical activity to identify epilepsy, migraine-related changes, or other electrical disorders Cleveland Clinic.Visual Evoked Potentials (VEP)
Measures the response of the visual cortex to light flashes, testing the optic pathway’s integrity Cleveland Clinic.Somatosensory Evoked Potentials (SEP)
Electrical stimulation of a peripheral nerve records brain responses, assessing the touch pathway up to the cortex Wikipedia.Brainstem Auditory Evoked Response (BAER)
Sounds are played into the ear while scalp electrodes record brainstem activity to test the auditory pathway Cleveland Clinic.
Imaging Tests
Magnetic Resonance Imaging (MRI)
Detailed pictures of brain structures identify tumors, stroke, or inflammation linked to AIWS Cleveland Clinic.Computed Tomography (CT) Scan
Rapid X-ray images reveal bleeding, tumors, or major structural issues that could trigger AIWS Cleveland Clinic.Functional MRI (fMRI)
Tracks changes in blood flow during tasks or at rest to map areas involved in sensory distortion Wikipedia.Single Photon Emission Computed Tomography (SPECT)
Uses a radioactive tracer to show areas of reduced or increased brain perfusion during AIWS episodes Wikipedia.
Non-Pharmacological Treatments
Below are evidence-based non-drug approaches, grouped into Physiotherapy & Electrotherapy, Exercise Therapies, Mind-Body Practices, and Educational/Self-Management strategies. Each entry includes its description, purpose, and mechanism.
A. Physiotherapy & Electrotherapy Therapies
Transcutaneous Electrical Nerve Stimulation (TENS)
Description: TENS delivers low-voltage electrical currents through skin electrodes to modulate pain.
Purpose: Alleviate headache pain and reduce attack frequency.
Mechanism: Stimulates large-diameter Aβ fibers at the spinal “gate,” inhibiting pain signals and inducing endorphin release Wikipedia.
Remote Electrical Neuromodulation (REN) Wearable (e.g., Nerivio®)
Description: A prescription armband device that delivers timed electrical pulses to the arm.
Purpose: Prevent and abort migraine-related AIWS episodes.
Mechanism: Activates conditioned pain modulation pathways, reducing central sensitization nerivio.com.
Repetitive Transcranial Magnetic Stimulation (rTMS)
Intramuscular Electrical Stimulation (IMES)
Description: Needle-based low-frequency electrical pulses to trigger points in neck muscles.
Purpose: Reduce muscle tension contributing to headache.
Mechanism: Modulates nociceptive input from cervical musculature, decreasing central sensitization PMC.
Vestibular Rehabilitation Therapy
Description: Exercises to improve balance and reduce dizziness.
Purpose: Address vertiginous symptoms sometimes accompanying AIWS.
Mechanism: Retrains vestibulo-ocular reflex and central adaptation to abnormal sensory input.
Ocular Motor Training
Description: Guided eye-tracking and coordination exercises.
Purpose: Enhance visual processing stability.
Mechanism: Strengthens oculomotor nerves, reducing visual perceptual errors.
Manual Cervical Traction
Description: Gentle hand-held pulling on the head to decompress cervical vertebrae.
Purpose: Relieve tension from neck structures that trigger headaches.
Mechanism: Creates intervertebral space, reducing nerve root irritation Healthline.
Mechanical Cervical Traction
Description: Device-assisted intermittent traction for the neck.
Purpose: Provide longer-duration decompression than manual techniques.
Mechanism: Cyclic stretching reduces muscle spasm and joint compression BioMed Central.
Galvanic Vestibular Stimulation
Description: Low-current delivered behind the ear to vestibular nerve.
Purpose: Normalize aberrant vestibular signals linked to perceptual distortions.
Mechanism: Modulates afferent vestibular pathways to recalibrate spatial perception.
Biofeedback (EMG-Based)
Description: Real-time muscle tension display via sensors and screen feedback.
Purpose: Teach voluntary control over neck and scalp muscle tension.
Mechanism: Trains relaxation responses, diminishing trigger-point activity Wikipedia.
B. Exercise Therapies
Aerobic Exercise
Description: Moderate-intensity activities (e.g., brisk walking) for ≥30 min/day.
Purpose: Reduce migraine frequency and AIWS triggers.
Mechanism: Releases endorphins, modulates stress hormones, and improves vascular tone Wikipedia.
Yoga & Stretching
Description: Postural and breathing exercises focusing on neck and shoulder flexibility.
Purpose: Alleviate musculoskeletal tension contributing to AIWS episodes.
Mechanism: Enhances parasympathetic activation, reduces sympathetic arousal.
Pilates
Description: Core-strengthening routines emphasizing posture.
Purpose: Promote spinal alignment and reduce cervicogenic triggers.
Mechanism: Improves core stability, decreasing compensatory neck strain.
Vestibular Gaze Stabilization Exercises
Description: Head and eye movements while focusing on a target.
Purpose: Enhance dynamic visual acuity and spatial orientation.
Mechanism: Strengthens vestibulo-ocular reflex pathways.
Progressive Resistance Training
Description: Gradual loading of neck and upper limb muscles.
Purpose: Increase muscular support of cervical spine, reducing trigger sensitivity.
Mechanism: Hypertrophies stabilizing muscles, lessening strain on triggers.
C. Mind-Body Therapies
Cognitive Behavioral Therapy (CBT)
Description: Psychological sessions targeting pain-related thoughts.
Purpose: Reduce anxiety and catastrophizing around AIWS episodes.
Mechanism: Reframes maladaptive cognitions, lowering stress-induced triggers Wikipedia.
Mindfulness Meditation
Description: Focused attention on present sensations without judgment.
Purpose: Enhance coping with sensory distortions.
Mechanism: Downregulates limbic reactivity, moderates cortical arousal.
Relaxation Training
Description: Techniques such as guided imagery and deep breathing.
Purpose: Reduce sympathetic overdrive contributing to migraines.
Mechanism: Activates parasympathetic pathways, decreasing noradrenergic tone.
D. Educational & Self-Management Strategies
Trigger Diary & Education
Description: Daily logging of symptoms, activities, diet, and sleep.
Purpose: Identify personal triggers and patterns.
Mechanism: Empowers behaviour modification to prevent episodes.
Patient Self-Management Workshops
Description: Structured group sessions teaching headache and AIWS strategies.
Purpose: Build skills in problem-solving, pacing, and stress reduction.
Mechanism: Enhances self-efficacy, reducing reliance on acute interventions.
Pharmacological Treatments
These medications target underlying migraine or epileptic mechanisms that precipitate AIWS. Each is listed with typical dosage, drug class, timing, and key side effects.
Topiramate (Anticonvulsant)
Dosage: Start 25 mg once daily, titrate to 50 mg twice daily for migraine prophylaxis NCBI.
Timing: Evening initiation, split doses AM/PM.
Side Effects: Paresthesia, cognitive slowing, weight loss.
Valproic Acid (Divalproex) (Anticonvulsant)
Dosage: 500–600 mg daily, targeting serum 21–50 µg/mL PubMed.
Timing: Usually split into two doses.
Side Effects: Nausea, tremor, weight gain, hepatotoxicity risk.
Propranolol (Beta-Blocker)
Dosage: 80–240 mg/day in divided doses for migraine prevention.
Timing: BID or TID.
Side Effects: Fatigue, bradycardia, exercise intolerance AAFP.
Amitriptyline (Tricyclic Antidepressant)
Dosage: 10–75 mg nightly.
Timing: Single dose at bedtime.
Side Effects: Sedation, dry mouth, weight gain AAFP.
Verapamil (Calcium Channel Blocker)
Dosage: 80–240 mg/day.
Timing: Divided doses.
Side Effects: Constipation, hypotension, edema.
Flunarizine (Calcium Channel Blocker; where available)
Dosage: 5–10 mg/day.
Timing: Bedtime.
Side Effects: Weight gain, depression.
Gabapentin (Anticonvulsant)
Dosage: 300 mg TID, titrating up to 1200 mg/day.
Timing: TID.
Side Effects: Dizziness, somnolence.
Lamotrigine (Anticonvulsant)
Dosage: Start 25 mg/day, up to 100–200 mg/day.
Timing: Single or divided dose.
Side Effects: Rash (rare Stevens-Johnson syndrome).
Sumatriptan (Triptan)
Dosage: 50–100 mg at headache onset.
Timing: May repeat after 2 hours once.
Side Effects: Paresthesia, chest tightness.
Botulinum Toxin A (Neurotoxin)
Dosage: 155–195 units IM across standard injection sites every 12 weeks.
Timing: Quarterly injections for chronic migraine.
Side Effects: Neck pain, muscle weakness, injection-site discomfort Wikipedia.
Dietary Molecular Supplements
Evidence supports certain nutraceuticals in migraine (and by extension AIWS) prevention.
Magnesium Oxide
Dosage: 400–600 mg/day orally.
Function: Modulates vascular tone and neurotransmission.
Mechanism: Inhibits cortical spreading depression, regulates glutamate release NCBIAmerican Migraine Foundation.
Riboflavin (Vitamin B₂)
Dosage: 400 mg/day for ≥3 months.
Function: Supports mitochondrial energy metabolism.
Mechanism: Enhances ATP production, stabilizing neuronal energy The Migraine TrustAmerican Academy of Neurology.
Coenzyme Q₁₀ (CoQ₁₀)
Dosage: 100–300 mg/day.
Function: Antioxidant supporting mitochondrial function.
Mechanism: Scavenges reactive oxygen species, modulates inflammatory cytokines PMCPractical Neurology.
Melatonin
Omega-3 Fatty Acids
Dosage: 1–2 g EPA/DHA daily.
Function: Anti-inflammatory effects.
Mechanism: Reduces production of pro-inflammatory eicosanoids.
Vitamin D₃
Dosage: 1,000–4,000 IU/day.
Function: Modulates immune and neuronal function.
Mechanism: May downregulate neuroinflammation.
Feverfew (Tanacetum parthenium)
Dosage: 50–100 mg of dried leaf extract daily.
Function: Traditional migraine prophylactic.
Mechanism: Inhibits serotonin release and prostaglandin synthesis.
Butterbur (Petasites hybridus)
Dosage: 25 mg twice daily, PA-free extract.
Function: Reduces migraine frequency.
Mechanism: Modulates inflammatory pathways; caution for hepatotoxic pyrrolizidine alkaloids.
Ginger (Zingiber officinale)
Dosage: 250 mg extract TID at onset.
Function: Acute nausea and pain relief.
Mechanism: Inhibits prostaglandin synthesis, reduces neurogenic inflammation.
L-Carnitine
Dosage: 500–1,000 mg/day, often combined with CoQ₁₀.
Function: Mitochondrial cofactor.
Mechanism: Facilitates fatty acid transport into mitochondria, supporting energy metabolism Migraine Canada.
Advanced/Experimental Therapies
Note: These are experimental and not standard for AIWS. They have been explored in other neurological or degenerative contexts.
Alendronate (Bisphosphonate)
Dosage: 70 mg once weekly (prevention of bone loss) Medscape Reference.
Function: Inhibits osteoclast-mediated bone resorption.
Mechanism: Encourages osteoclast apoptosis; proposed to modulate neuro-inflammatory mediators in experimental models.
Zoledronic Acid (Bisphosphonate)
Dosage: 5 mg IV once yearly.
Function: Potent inhibitor of bone resorption.
Mechanism: Similar to alendronate, with long-lasting effects on bone and inflammatory cytokines.
Platelet-Rich Plasma (Regenerative)
Dosage: Autologous PRP injections, volume varies.
Function: Delivers growth factors to injured neural tissue.
Mechanism: May promote neural repair via PDGF, TGF-β, VEGF release.
Nerve Growth Factor (Regenerative)
Dosage: Experimental—dose defined per trial.
Function: Supports neuronal survival and plasticity.
Mechanism: Binds TrkA receptors, promoting axonal growth.
Hyaluronic Acid Injection (Viscosupplementation)
Dosage: 2 mL intra-articular monthly (e.g., knee OA).
Function: Lubricates joints; theorized to modulate nociceptive input.
Mechanism: Enhances extracellular matrix, dampening mechanoreceptor-driven pain signals.
Mesenchymal Stem Cell Therapy
Dosage: 1–10 × 10⁶ cells/kg IV or local injection.
Function: Potential to regenerate damaged neural circuits.
Mechanism: Paracrine release of neurotrophic factors, immunomodulation.
Surgical Procedures
While AIWS itself requires no surgery, addressing refractory migraine triggers may help:
Supraorbital & Supratrochlear Nerve Decompression
Procedure: Endoscopic removal of corrugator muscle or nerve decompression in glabellar region.
Benefits: Significant reduction in chronic migraine pain in well-selected patients Wikipedia.
Temporal Branch Decompression
Procedure: Endoscopic muscle resection or nerve branch transection in temporal area.
Benefits: Decreases temporal migraine frequency and intensity.
Occipital Nerve Decompression
Procedure: Resection of semispinalis capitis muscle segment around greater occipital nerve.
Benefits: Relief in occipital migraines, improved quality of life.
Arterial Cauterization (Shevel Procedure)
Procedure: Cauterize dilated scalp vessels (external carotid branches) identified as pain sources.
Benefits: Long-term migraine prevention; up to 29% complete elimination at 5 years Wikipedia.
Migraine Trigger Site Surgery
Procedure: Combination of muscle resection, nerve decompression, and vessel cauterization at multiple trigger sites.
Benefits: Up to 88% positive response, with significant symptom reduction Wikipedia.
Preventive Strategies
Adequate Hydration: ≥2 L/day to prevent dehydration-triggered episodes.
Regular Sleep Schedule: Maintain consistent sleep–wake times.
Balanced Diet: Avoid tyramine-rich foods; eat small, frequent meals.
Stress Management: Daily relaxation or mindfulness practice.
Screen Breaks: Limit continuous screen exposure to reduce visual strain.
Environmental Controls: Use soft lighting and avoid flickering lights.
Ergonomic Posture: Optimize desk/chair setup to reduce neck tension.
Regular Exercise: Moderate aerobic activity most days Wikipedia.
Trigger Identification: Use a diary to eliminate personal triggers.
Routine Medical Check-ups: Monitor comorbid conditions (e.g., anemia, hypertension).
When to See a Doctor
First Episode: Any new perceptual distortion requires evaluation.
Frequent Episodes: ≥2 AIWS episodes/month despite self-care.
Severe Symptoms: Hallucinations, disorientation, or prolonged episodes >1 hour.
Neurological Signs: Weakness, incoordination, or altered consciousness.
Infection Signs: Fever or neck stiffness suggesting encephalitis.
What-To-Do & What-To-Avoid Tips
Do:
Keep a detailed symptom diary.
Engage in regular relaxation or meditation.
Stay physically active within tolerance.
Maintain hydration and balanced meals.
Use prescribed migraine prophylaxis consistently.
Avoid:
6. Skipping meals or fasting.
7. Excessive caffeine or alcohol intake.
8. Irregular sleep patterns or “catch-up” weekends.
9. Over-reliance on acute painkillers (risk of rebound).
10. Prolonged screen time without breaks.
Frequently Asked Questions (FAQs)
What exactly causes AIWS?
AIWS arises from transient dysfunction in brain regions processing perception—often linked to migraines, infections, epilepsy, or rarely brain lesions Wikipedia.Is AIWS permanent?
No—AIWS is typically self-limiting. Episodes last minutes to an hour, with full resolution in most cases.Can adults develop AIWS?
Yes, though it’s more common in children and adolescents, adults can experience AIWS, especially when associated with migraine or infections.Are AIWS episodes dangerous?
The distortions themselves aren’t harmful, but reassurance and evaluation are vital to exclude serious causes.How is AIWS diagnosed?
Diagnosis is clinical, based on history of episodic perceptual distortions and exclusion of other neurological conditions via imaging and EEG.Can lifestyle changes help prevent AIWS?
Yes—consistent sleep, stress management, hydration, and trigger avoidance can reduce episode frequency.What role do migraines play?
Migraines are the most common AIWS trigger; effective migraine prophylaxis often reduces AIWS episodes Cleveland Clinic.Are there specific medications for AIWS?
No AIWS-specific drug exists; treatment targets underlying causes (e.g., anticonvulsants for epilepsy, triptans or prophylactics for migraine).Can therapy like CBT help?
Yes—CBT can improve coping strategies and reduce stress-mediated triggers Wikipedia.Should I avoid screen time?
Limiting prolonged screen exposure and taking regular breaks helps prevent eye strain and potential triggers.Is there a cure for AIWS?
There’s no cure; management focuses on controlling underlying conditions and preventive measures.Can infections cause AIWS?
Epstein–Barr virus and other viral encephalitides are known infectious triggers, particularly in children.Is AIWS linked to mental illness?
No—though distortions may resemble hallucinations, AIWS is neurological and episodic, not psychiatric.When is imaging needed?
Brain MRI/EEG is indicated if atypical features, prolonged symptoms, or focal neurological signs are present.Can AIWS recur later in life?
While most cases resolve in adolescence, recurrent episodes can occur if underlying triggers persist.
Disclaimer: Each person’s journey is unique, treatment plan, life style, food habit, hormonal condition, immune system, chronic disease condition, geological location, weather and previous medical history is also unique. So always seek the best advice from a qualified medical professional or health care provider before trying any treatments to ensure to find out the best plan for you. This guide is for general information and educational purposes only. Regular check-ups and awareness can help to manage and prevent complications associated with these diseases conditions. If you or someone are suffering from this disease condition bookmark this website or share with someone who might find it useful! Boost your knowledge and stay ahead in your health journey. We always try to ensure that the content is regularly updated to reflect the latest medical research and treatment options. Thank you for giving your valuable time to read the article.
The article is written by Team RxHarun and reviewed by the Rx Editorial Board Members
Last Updated: July 12, 2025.
