Non-contained thecal sac indentation refers to a condition in which disc material that has broken through (extruded) the annulus fibrosus—without being held in place by the annular fibers or posterior longitudinal ligament—pushes directly against and indents the thecal sac, the dural sheath enclosing the spinal cord and cauda equina. In non-contained herniations, this free or partially attached disc fragment can create focal pressure on the thecal sac, potentially reducing cerebrospinal fluid space and irritating neural elements SpineInfoRadiopaedia.


Anatomy of the Thecal Sac

The thecal (dural) sac is a tough, membranous tube of dura mater that contains and protects the spinal cord, cauda equina, and cerebrospinal fluid (CSF).

  • Structure & Location
    The dura forms a continuous sheath from the cranial foramen magnum down to the second sacral vertebra (S2), at which point it tapers over the filum terminale. Within the vertebral (spinal) canal, the sac is separated from bone by the epidural space filled with fat and venous plexuses Wikipedia.

  • Attachments (Origin & “Insertion”)
    Cranially, the dural sac adheres firmly at the foramen magnum to the cranial dura. Caudally, it narrows at S2 to envelop the filum terminale, anchoring the terminal end of the spinal cord. Along its length, dural root sleeves project laterally to form protective sheaths around exiting spinal nerves Wikipedia.

  • Blood Supply
    The spinal dura receives small arterial branches (meningeal arteries) and venous drainage via meningeal veins. These vessels run with the radicular arteries and veins through the intervertebral foramina into the epidural space KenhubNCBI.

  • Nerve Supply
    Sensory fibers from the meningeal branches of each spinal (segmental) nerve innervate the spinal dura, carrying pain and proprioceptive input from the thecal sac Radiopaedia.

  • Functions

    1. Protection: Shields the spinal cord and nerve roots from external forces.

    2. CSF Containment: Maintains the reservoir of cerebrospinal fluid, providing nutrients and buoyancy.

    3. Shock Absorption: Acts as a fluid-filled cushion against mechanical impacts.

    4. Neural Conduit: Durally encases nerve root sleeves, guiding roots to their foraminal exits.

    5. Therapeutic Access: Serves as the route for lumbar puncture (intrathecal injections) and epidural anesthesia.

    6. Structural Stabilization: Anchors the spinal cord via attachments at cranial and sacral ends.


Types of Non-Contained Thecal Sac Indentation

Non-contained thecal sac indentation arises chiefly from two forms of disc herniation, which may also migrate within the epidural space:

  1. Disc Extrusion – A herniated disc with a narrow neck and a broad apex extending beyond the annulus fibrosus, still connected to the parent disc but lacking containment by annular fibers or ligament, indenting the thecal sac Radiology Assistant.

  2. Disc Sequestration – A free fragment completely detached from the parent disc that migrates in the epidural space, potentially moving to contact and indent the thecal sac Radiology Assistant.

  3. Migrated Extruded Fragment – An extrusion that has shifted cranially or caudally within the canal before contacting the thecal sac Radiopaedia.


Causes

Non-contained indentation of the thecal sac most commonly results from disc material breaching its natural barriers, but several factors predispose to this process:

  1. Degenerative Disc Disease: Age-related breakdown of disc structure leading to annular tears NCBI.

  2. Acute Trauma: Sudden axial loading (falls, motor vehicle collisions) causing extrusion Spine-health.

  3. Repetitive Microtrauma: Chronic bending or lifting strains Spine-health.

  4. Genetic Predisposition: Genetic variations affecting collagen and disc matrix Spine-health.

  5. Advanced Age: Incidence peaks in 30–50 year olds Spine-health.

  6. Obesity (BMI > 30): Increases mechanical disc loading and inflammation Spine-health.

  7. Smoking (Nicotine): Impairs disc nutrition and healing Spine-health.

  8. Occupation: Heavy manual labor, prolonged sitting Spine-health.

  9. Poor Posture: Sustained flexed or rotated spine positions PMC.

  10. Sedentary Lifestyle: Weak core & paraspinal muscles Spine-health.

  11. High-Impact Sports: Forces beyond disc tolerance Spine-health.

  12. Vertebral Endplate Damage: Vertical herniation through endplate fractures Spine-health.

  13. Connective Tissue Disorders: Ehlers-Danlos, Marfan syndromes causing annular weakness Spine-health.

  14. Diabetes Mellitus: Promotes glycation and disc degeneration PMC.

  15. Previous Disc Surgery: Annular defects increase re-herniation risk PubMed.

  16. Spinal Stenosis: Osteophytes or ligamentum flavum hypertrophy may tear annulus secondarily SpineInfo.

  17. Synovial Cysts: Facet joint cysts can erode or tear annulus Radiopaedia.

  18. Epidural Lipomatosis: Excess fat may cause annular stress SpineInfo.

  19. Vibration Exposure: Machinery or vehicle vibrations weaken discs PMC.

  20. Congenital Disc Weakness: Short pedicles or inherent annular defects NCBI.


Symptoms

When non-contained disc material indents the thecal sac, symptoms arise from direct pressure on neural tissues and disrupted CSF flow SpineInfo:

  1. Localized back pain

  2. Radicular leg pain (sciatica)

  3. Buttock or thigh discomfort

  4. Calf or foot pain

  5. Numbness along a dermatomal pattern

  6. Paresthesia (“tingling” or “pins and needles”)

  7. Muscle weakness in affected myotomes

  8. Decreased reflexes in the leg

  9. Muscle cramps or spasms

  10. Gait instability

  11. Clonus or hyperreflexia (if higher level)

  12. Lhermitte’s sign (electric shock sensation)

  13. Saddle anesthesia (perineal numbness)

  14. Bladder urgency or retention

  15. Bowel incontinence or constipation

  16. Sexual dysfunction (erectile issues)

  17. Foot drop (difficulty dorsiflexing)

  18. Loss of proprioception in lower limbs

  19. Cold or burning sensations

  20. Rest pain (worsened at night)


Diagnostic Tests

Accurate diagnosis combines history, exam, and imaging/electrophysiology NCBI:

  1. Magnetic Resonance Imaging (MRI) – Gold standard for soft tissue.

  2. Computed Tomography (CT) – Better for bony detail.

  3. CT Myelogram – CT plus contrast in CSF; shows canal obstruction.

  4. X-ray (Flexion/Extension Views) – Screens for instability.

  5. Discography – Provocative test with intradiscal contrast.

  6. Electromyography (EMG) – Assesses nerve root irritation.

  7. Nerve Conduction Study (NCS) – Evaluates peripheral nerve function.

  8. Somatosensory Evoked Potentials (SSEPs) – Spinal cord conduction.

  9. Diffusion Tensor Imaging (DTI) – Microstructural nerve changes.

  10. Myelography – Contrast in subarachnoid space, visualizing indentation.

  11. Bone Scan (SPECT) – Detects active bone pathology.

  12. Lumbar Puncture (CSF Analysis) – Rarely for infection/bleed.

  13. Ultrasound-Guided Injections – Diagnostic or therapeutic.

  14. Dynamic Fluoroscopy – Real-time movement assessment.

  15. Computed Tomographic Epidurography – Epidural space imaging.

  16. Blood Tests – Inflammatory markers (ESR, CRP) for abscess.

  17. Genetic Testing – For connective tissue disorders.

  18. Somatosensory Evoked Response Testing – Spinal cord functional study.

  19. Vertebral Venography – Rare, for vascular causes.

  20. Quantitative Sensory Testing – Sensory nerve function.


Non-Pharmacological Treatments

Conservative care is first-line in most cases SpineInfo:

  1. Physical therapy (core stabilization)

  2. McKenzie extension exercises

  3. Flexion/distraction therapy

  4. Transcutaneous electrical nerve stimulation (TENS)

  5. Heat and cold modalities

  6. Manual therapy (mobilization/manipulation)

  7. Stretching routines

  8. Yoga and Pilates

  9. Acupuncture

  10. Massage therapy

  11. Spinal traction

  12. Inversion therapy

  13. Ergonomic optimization (workstation)

  14. Posture training

  15. Activity modification (avoiding aggravating movements)

  16. Weight management

  17. Water therapy (aquatic exercises)

  18. Cognitive behavioral therapy (pain coping)

  19. Neural mobilization techniques

  20. Kinesio taping

  21. Bracing (lumbar support belts)

  22. Chiropractic adjustments

  23. Disc decompression therapy

  24. Core muscle strengthening

  25. Biofeedback

  26. Education on body mechanics

  27. Occupational therapy

  28. Mind-body relaxation techniques

  29. Spinal stabilization devices

  30. Lifestyle counseling (smoking cessation, nutrition)


Drugs

When needed, medications can target pain and inflammation SpineInfo:

  1. Ibuprofen (NSAID)

  2. Naproxen (NSAID)

  3. Acetaminophen

  4. Celecoxib (COX-2 inhibitor)

  5. Ketorolac (injectable NSAID)

  6. Cyclobenzaprine (muscle relaxant)

  7. Tizanidine (muscle relaxant)

  8. Gabapentin (anticonvulsant)

  9. Pregabalin

  10. Amitriptyline (TCA)

  11. Duloxetine (SNRI)

  12. Tramadol (opioid-like analgesic)

  13. Oxycodone (opioid)

  14. Morphine (opioid)

  15. Lidocaine patch (topical)

  16. Capsaicin cream (topical)

  17. Prednisone (oral corticosteroid)

  18. Dexamethasone (injectable steroid)

  19. Epidural steroid injection (ESI)

  20. Botulinum toxin (off-label for spasm)


Surgeries

Reserved for severe or refractory cases SpineInfo:

  1. Microdiscectomy (mini-open)

  2. Endoscopic discectomy

  3. Laminectomy (decompression)

  4. Laminotomy (partial)

  5. Foraminotomy (nerve-root decompression)

  6. Posterior lumbar interbody fusion (PLIF)

  7. Transforaminal lumbar interbody fusion (TLIF)

  8. Anterior lumbar interbody fusion (ALIF)

  9. Total disc replacement (arthroplasty)

  10. Dynamic stabilization systems


Preventions

Protect your spine and reduce risk Spine-health:

  1. Maintain healthy weight

  2. Regular exercise (aerobic, strength)

  3. Proper lifting (bend knees, keep load close)

  4. Ergonomic workstations

  5. Frequent posture breaks (avoid prolonged sitting)

  6. Core strengthening routines

  7. Quit smoking

  8. Stay hydrated (disc nutrition)

  9. Flexibility training (hamstrings, hip flexors)

  10. Load management (avoid sudden heavy lifts)


When to See a Doctor

Seek prompt medical evaluation if you experience:

  • Progressive weakness or difficulty walking

  • Loss of bladder or bowel control

  • Saddle anesthesia (numbness in groin)

  • Severe, unrelenting pain not improved by rest

  • Significant sensory changes (numbness, tingling)

  • Fever with back pain (suggests infection)

  • Night pain awakening you

  • Post-surgical recurrence of symptoms


Frequently Asked Questions (FAQs)

  1. What exactly is non-contained thecal sac indentation?
    It’s when disc material that has broken free presses directly on the dural sac, indenting its surface.

  2. How does it differ from contained herniation?
    Contained herniations stay within annular fibers; non-contained ones break through and can move freely.

  3. What symptoms suggest nerve root involvement?
    Radiating pain, numbness, tingling, or weakness along a specific nerve distribution (dermatome).

  4. Why is MRI preferred for diagnosis?
    MRI best visualizes soft tissue details, including disc fragments and CSF spaces.

  5. Can non-surgical care really work?
    Yes—up to 90% improve with physical therapy, activity modification, and pain management.

  6. When is surgery necessary?
    If there’s progressive neurological deficit, cauda equina signs, or intractable pain despite 6–12 weeks of care.

  7. Is this condition permanent?
    Many patients recover fully; free fragments can resorb over months.

  8. What exercises help recovery?
    Core stabilization, McKenzie extension, and gentle stretching.

  9. Can I return to work?
    Often yes—with ergonomic adjustments and gradual activity progression.

  10. Are there long-term complications?
    Rarely—most regain function; recurrent herniation occurs in ~5–15%.

  11. Does age affect recovery?
    Older patients may take slightly longer but still respond well to treatment.

  12. Is epidural steroid injection safe?
    Generally yes, though risks include infection, bleeding, and transient headache.

  13. What lifestyle changes reduce recurrence?
    Weight control, smoking cessation, regular exercise, and safe lifting habits.

  14. Can I play sports again?
    After appropriate rehabilitation, many resume non-contact sports safely.

  15. How long until I see improvement?
    Symptoms often start improving within 4–6 weeks of conservative management.

Disclaimer: Each person’s journey is unique, treatment plan, life style, food habit, hormonal condition, immune system, chronic disease condition, geological location, weather and previous medical  history is also unique. So always seek the best advice from a qualified medical professional or health care provider before trying any treatments to ensure to find out the best plan for you. This guide is for general information and educational purposes only. Regular check-ups and awareness can help to manage and prevent complications associated with these diseases conditions. If you or someone are suffering from this disease condition bookmark this website or share with someone who might find it useful! Boost your knowledge and stay ahead in your health journey. We always try to ensure that the content is regularly updated to reflect the latest medical research and treatment options. Thank you for giving your valuable time to read the article.

The article is written by Team Rxharun and reviewed by the Rx Editorial Board Members

Last Updated: May 03, 2025.

      RxHarun
      Logo
      Register New Account