Lacrimal Gland Prolapse

Your lacrimal glands make tears. Each eye has one, sitting up and out toward the eyebrow in a small bony pocket called the “lacrimal fossa.” A web of thin tissues (ligaments and eyelid muscle attachments) holds the gland up and back where it belongs. Lacrimal gland prolapse (also called lacrimal gland ptosis) means that this support has loosened or been pulled away, so the gland sags forward and downward. From the outside, this often looks like a puffy bulge in the outer part of the upper eyelid, especially when you look down or when someone gently lifts the upper lid. The bulge itself is usually soft and can often be pushed back temporarily with a fingertip.

Why it matters: a prolapsed gland can cause cosmetic asymmetry, heaviness, mechanical droop of the outer eyelid, and sometimes watering (tearing) or dryness because the gland is slightly out of position. In most cases it is benign (not cancer). But sometimes inflammation or, rarely, a tumor can push the gland forward and mimic prolapse. That is why a careful exam—and imaging when needed—is important.

Your main tear gland (the lacrimal gland) sits in a small “shelf” of bone at the upper-outer corner of each eye socket. In lacrimal gland prolapse, that gland slips forward and downward out of its natural pocket. From the outside, it usually looks like a soft, painless bulge at the outer part of the upper eyelid. The bulge can be more obvious when you look down or when the lid is lifted. The gland itself is usually healthy; it’s just out of place. EyeWikiBioMed Central

Think of the lacrimal gland as a small, soft almond in a hammock. With age, rubbing, prior surgery, or inflammation, the “hammock strings” (the lateral horn of the levator aponeurosis, Whitnall’s ligament, and nearby connective tissues) get stretched, thinned, or torn. The gland then slips out of its nook and sits lower and more forward, poking the outer eyelid from behind. Gravity makes it show more when you look down. If the gland is swollen (from infection, autoimmune disease, or a mass), it takes up more space and pushes forward, too.

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Types

1. Primary (age-related) prolapse
   The most common type. The gland sags because the natural supports get weaker with age. No infection or tumor is present.

2. Secondary to surgery
   After upper-eyelid surgery (for excess skin or for droopy lid repair), scarring or altered support can let the gland slide forward.

3. Secondary to trauma
   A blow to the brow or orbit can tear the tissue that holds the gland, allowing it to drop.

4. Inflammatory “pseudoprolapse”
   The gland is swollen from infection or inflammation (for example, dacryoadenitis), so it looks prolapsed even if the supports are intact. When swelling settles, the bulge may lessen.

5. Tumor-associated forward displacement
   A benign or malignant mass in or near the gland pushes it outward. This must be ruled out if the bulge is firm, progressive, painful, or unequal.

6. Congenital laxity (rare)
   Some people are born with looser connective tissue or a gland that sits lower, so a bulge is present very early in life.

7. Syndromic/connective tissue–related
   Conditions that make body tissues looser (for example, certain inherited connective tissue disorders) can contribute to prolapse.

8. Intermittent or positional prolapse
   The bulge becomes more obvious with looking down, straining, allergy flares, or fluid retention, then looks smaller at other times.

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Causes

1. Age-related thinning of eyelid supports
   With time, the “strings” that hold the gland (lateral horn of the levator, Whitnall’s ligament, and fascia) stretch. This is the most common reason the gland slips forward.

2. General eyelid laxity (dermatochalasis)
   Extra upper-lid skin and lax tissues reduce the counter-pressure that keeps the gland tucked in place, allowing a soft outer-lid bulge.

3. Chronic eye rubbing
   Rubbing pushes on the outer upper lid again and again, loosening the supports and nudging the gland forward.

4. Prior upper-eyelid blepharoplasty
   Cosmetic removal of skin/fat can disturb the delicate attachments, leaving the gland less supported and more likely to prolapse.

5. Prior ptosis (droopy-lid) repair
   Surgery on the levator aponeurosis can change the lateral horn’s tension; in some cases that lets the gland slide outward.

6. Orbital or brow trauma
   A hit or fracture near the lacrimal fossa can tear tissues that hold the gland, producing a new asymmetric bulge afterward.

7. Acute bacterial dacryoadenitis
   Infection makes the gland swollen and tender. The swollen gland pushes forward—sometimes mistaken for true prolapse.

8. Viral dacryoadenitis (e.g., mumps, EBV)
   Viral swelling expands the gland quickly, making a sudden outer-lid lump that often improves as the virus resolves.

9. Autoimmune dacryoadenitis (Sjögren’s syndrome)
   Immune cells attack the lacrimal gland, causing enlargement, soreness, and reduced tear quality—bulging can result.

10. IgG4-related ophthalmic disease
    IgG4-positive plasma cells infiltrate the gland, making it firm and enlarged, which pushes it forward.

11. Sarcoidosis
    Granulomas form in the gland, enlarging it and displacing it, often with other body signs like cough or skin lesions.

12. Thyroid eye disease (orbitopathy)
    Inflammation and fat/muscle swelling in the orbit can shift tissues and secondarily allow the gland to sit more anteriorly.

13. Benign lacrimal gland tumor (pleomorphic adenoma)
    A slow, firm mass in the gland gradually presses it outward; typically painless but steadily enlarging.

14. Malignant lacrimal gland tumor (e.g., adenoid cystic carcinoma)
    Pain, rapid change, and firmness are red flags; the mass pushes the gland and nearby tissues forward.

15. Lacrimal gland lymphoma
    A rubbery, often painless enlargement. It can cause a smooth bulge and needs biopsy confirmation.

16. Metastatic disease to the gland (rare)
    Cancers like breast cancer can spread to orbital tissues and alter gland position.

17. Connective tissue disorders (e.g., Ehlers–Danlos)
    Naturally looser collagen means weaker “hammock strings,” so the gland is more likely to sag.

18. Chronic allergy and eyelid edema
    Long-standing swelling and rubbing loosen support, and fluid retention can make the bulge look larger on bad days.

19. Obesity with orbital fat prolapse
    Extra orbital fat can crowd the superolateral space and nudge the gland forward.

20. Long-term corticosteroid exposure
    Steroids thin connective tissue over time, making supports weaker and increasing the chance of prolapse.

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Symptoms

1. Outer upper-lid bulge
   A soft, rounded lump near the outer eyelid, more obvious when looking down or when the lid is lifted.

2. Heaviness and fullness
   The eyelid can feel weighty, like something is “in the way” at the outer corner.

3. Cosmetic asymmetry
   One eye looks puffy or different, which bothers many people even if it does not hurt.

4. Mechanical droop (lateral ptosis)
   The outer lid can hang slightly lower, narrowing the eye opening on that side.

5. Tearing (epiphora)
   The malpositioned gland or reflex irritation can lead to watery eyes, especially outdoors or in wind.

6. Dry-eye sensations
   Ironically, some people feel dryness, burning, or grittiness, because tear composition and spread are disturbed.

7. Tenderness with inflammation
   If the gland is inflamed, pressing on the area can hurt; there may be warmth and redness.

8. Foreign-body feeling on blinking
   The bulge changes how the lid slides on the eye, so blinking can feel scratchy.

9. Redness of the outer conjunctiva
   Mild, persistent redness near the outer corner can accompany the bulge.

10. Worse at day’s end or when looking down
    Gravity, fatigue, and fluid shifts make the bulge more visible later in the day or in downgaze.

11. Intermittent swelling with colds/allergy
    The bulge looks bigger during allergy flares or upper-respiratory infections and shrinks afterward.

12. Upper-field shadowing
    A droopy outer lid can slightly block the upper-outer part of the vision field.

13. Pressure or ache near the eyebrow
    A dull fullness or pressure can be felt, especially if the gland is inflamed or the tissues are stretched.

14. Contact-lens discomfort
    Tear film instability and lid mechanics can make lenses feel dry or irritating.

15. Anxiety about a “lump”
    The look of a lump is worrying; reassurance or further testing may be needed to rule out serious causes.

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Diagnostic Tests

Not every test below is needed for every person. Doctors start with a careful exam, then add tests only if something looks atypical, painful, progressive, or unequal, or if there are systemic symptoms (fever, weight loss, dry mouth, cough, etc.).

A) Physical Examination

1. Visual inspection in different gaze positions
   The clinician looks straight ahead, up, and down. A true lacrimal gland prolapse typically bulges more in downgaze and sits at the outer upper lid. Symmetry between the two eyes is compared.

2. Palpation of the superolateral upper lid
   Gentle fingertip pressure feels a soft, mobile mound that can often be nudged back temporarily. Increased warmth or tenderness suggests inflammation rather than simple age-related sag.

3. Upper-lid eversion and fornix inspection
   Flipping the lid exposes the palpebral lobe and the area where tear ducts open. Seeing the gland’s edge there and any discharge helps confirm a gland-related bulge instead of a skin cyst or fat pad.

4. Schirmer I tear test (without anesthesia)
   A small paper strip rests at the outer lower lid for 5 minutes to measure tear production. Low wetting suggests tear-gland dysfunction (e.g., Sjögren’s), which can enlarge or displace the gland.

5. Eyelid measurements (MRD1) and levator function
   The margin-reflex distance (how high the lid sits) and the lid’s lifting power are measured. Lateral (outer) droop with otherwise normal levator function supports prolapse rather than classic muscular ptosis.

B) Manual/Bedside Maneuvers

6. Retropulsion test
   The examiner gently pushes the globe backward through the closed lid. If a soft structure slides back in the outer upper lid, it supports the idea of a mobile gland rather than a fixed hard mass.

7. Digital reduction (“push-back”) test
   With clean fingers and very light pressure, the bulge is pushed superolaterally. Temporary reduction of the lump that reappears after release is typical of prolapse.

8. Valsalva or strain provocation
   Brief gentle strain (like exhaling against a closed mouth) or head-down positioning can increase venous congestion, making a soft prolapse more visible. A mass usually does not change with this.

9. Lacrimal duct expression/inspection
   Gentle pressure over the gland while watching the outer upper conjunctiva can show clear tear secretion. Pus or stringy mucus suggests active inflammation (dacryoadenitis).

C) Laboratory and Pathological Tests

10. Complete blood count (CBC)
    Looks for infection (high white cells) or blood changes seen with some lymphomas. Normal results are common in simple prolapse.

11. ESR and CRP (inflammation markers)
    Elevated levels support active inflammation (infection or autoimmune). Normal levels fit better with age-related sag.

12. Autoimmune antibody panel (ANA, anti-SSA/Ro, anti-SSB/La)
    Helpful if there are dry-mouth/dry-eye symptoms or other autoimmune signs. A positive result points toward Sjögren’s, which can enlarge and stiffen the gland.

13. Serum IgG4 level
    Elevated IgG4 suggests IgG4-related disease, which commonly involves the lacrimal gland and can mimic tumors.

14. Biopsy with histopathology (incisional or core needle, when indicated)
    If the bulge is firm, rapidly growing, painful, or asymmetric, a sample is taken. Microscopy and immunostains distinguish inflammation (including IgG4), lymphoma, and epithelial tumors like pleomorphic adenoma or adenoid cystic carcinoma.

D) Electrodiagnostic Tests

15. Visual evoked potential (VEP)
    If imaging or symptoms suggest the optic nerve might be affected by an orbital mass (not typical of simple prolapse), VEP checks whether signals from the eye reach the brain normally.

16. Orbicularis oculi EMG / blink-reflex testing
    Used only if there is concern about facial nerve function (for example, unusual lid closure weakness). This helps separate nerve problems from mechanical eyelid issues.

E) Imaging Tests

17. Orbital ultrasound (B-scan)
    A quick, radiation-free scan that can show a soft, anteriorly positioned gland and help detect solid masses or cysts.

18. CT scan of the orbits
    Shows the bony lacrimal fossa and the gland’s location. CT is excellent for calcification, bone change, and overall anatomy, and it can reveal a mass pushing the gland forward.

19. MRI of the orbits with contrast (± diffusion sequences)
    Best for soft-tissue detail. MRI shows whether the gland is simply ptotic (sagged), inflamed, or infiltrated by a tumor. Diffusion-weighted images help differentiate lymphoma from inflammation.

20. Systemic staging imaging when red flags exist (e.g., PET-CT or chest CT/X-ray)
    If biopsy or labs suggest lymphoma, IgG4 disease, sarcoidosis, or metastasis, body imaging looks for other involved sites to guide treatment.

Non-pharmacological treatments

(These improve comfort/appearance or reduce triggers; they don’t “push” the gland back for good. Lasting correction is surgical—see surgery section.)

1. Watchful waiting. If the bulge is small, painless, and you’re okay with how it looks, simply monitor.
   Purpose: Avoid unnecessary treatment. Mechanism: Many cases are stable for years.

2. Allergy control. Close windows in high-pollen times; use air filters; wash pillowcases often.
   Purpose: Reduce swelling and rubbing that worsen prolapse. Mechanism: Less histamine-driven lid edema.

3. No eye rubbing. Use a clean tissue or chilled artificial tears instead.
   Purpose: Protect stretched supports. Mechanism: Less mechanical strain.

4. Cold compress during flares.
   Purpose: Shrink temporary swelling. Mechanism: Vasoconstriction reduces tissue edema.

5. Moisture-chamber glasses or sleep shields (especially in AC or windy settings).
   Purpose: Keep the ocular surface moist. Mechanism: Reduces evaporation—an evidence-based dry-eye tactic. tfosdewsreport.org+1

6. Blink breaks & screen hygiene. 20-20-20 rule; place monitors slightly below eye level.
   Purpose: Reduce dryness/irritation. Mechanism: Improves tear spread.

7. Humidifier at home/office.
   Purpose: Add moisture to air. Mechanism: Less tear evaporation.

8. Lid hygiene (gentle). Micellar lid cleansers; avoid harsh scrubbing.
   Purpose: Calm lid margins if gritty. Mechanism: Reduces biofilm/inflammation.

9. Sleep with head slightly elevated.
   Purpose: Reduce morning lid swelling. Mechanism: Less overnight venous pooling.

10. Limit dietary salt in the evening.
    Purpose: Less puffiness next morning. Mechanism: Decreases fluid retention.

11. Quit smoking.
    Purpose: Protect collagen and reduce TED risk. Mechanism: Smoking worsens thyroid eye disease activity. PMC

12. UV-blocking sunglasses outdoors.
    Purpose: Reduce irritation. Mechanism: Shields from wind/UV.

13. Temporary cosmetic camouflage.
    Purpose: Hide mild bulge (makeup/hairstyle). Mechanism: Non-medical coping.

14. Contact lens time-out if symptomatic.
    Purpose: Avoid friction over bulge. Mechanism: Reduces mechanical irritation.

15. Weight management and exercise.
    Purpose: Lower edema/inflammation. Mechanism: Systemic anti-inflammatory benefits.

16. Manage sinus/allergy triggers.
    Purpose: Fewer flares. Mechanism: Less adjacent tissue swelling.

17. Treat coexisting dry eye with non-drug measures (preservative-free tears, warm compresses for MGD).
    Purpose: Comfort. Mechanism: Follows TFOS DEWS staged care. PubMed

18. Scleral or moisture-retaining lenses in select severe dry eye.
    Purpose: Protect the cornea. Mechanism: Fluid reservoir over the eye (specialist-fit). PubMed

19. Therapeutic taping at night (rare, short-term).
    Purpose: Reduce rubbing if you touch lids during sleep. Mechanism: Physical reminder; use only if advised.

20. Plan surgery at a calm, stable phase (if you want definitive correction).
    Purpose: Better, longer-lasting results. Mechanism: Operating when inflammation/allergy is quiet leads to cleaner planes and healing.

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Medication options

Key point: Medicines do not “fix” a prolapsed gland. They treat symptoms (dryness, irritation) or associated conditions (allergy, inflammation, infection). Doses below are typical ranges—your doctor may change them based on you.

1. Preservative-free artificial tears (e.g., carboxymethylcellulose 0.5%)
   Dose: 1 drop, 3–6×/day; more often if needed. Purpose: Comfort, reduce friction. Mechanism: Replaces and stabilizes the tear film. PubMed

2. Lubricating ointment (petrolatum/mineral oil) at bedtime
   Dose: ¼-inch ribbon nightly. Purpose: Nighttime protection. Mechanism: Slows evaporation. PubMed

3. Topical antihistamine/mast-cell stabilizers (e.g., olopatadine, ketotifen)
   Dose: 1 drop 1–2×/day during allergy season. Purpose: Itch control; less rubbing. Mechanism: Blocks histamine; stabilizes mast cells.

4. Oral non-sedating antihistamines (e.g., cetirizine 10 mg daily)
   Purpose: Systemic allergy control. Mechanism: Reduces histamine effects; note possible extra dryness.

5. Short course low-potency steroid drops (e.g., loteprednol 0.5% QID for 1–2 weeks, doctor-directed)
   Purpose: Calm significant inflammatory flares. Mechanism: Anti-inflammatory; risk: pressure rise/cataract if misused—must be supervised. PubMed

6. Topical calcineurin inhibitors for eyelid skin (e.g., tacrolimus 0.03% ointment nightly for dermatitis)
   Purpose: Treat allergic/atopic lid eczema, reduce rubbing. Mechanism: Local immune modulation.

7. Cyclosporine A eye drops 0.05–0.1% (twice daily)
   Purpose: Chronic inflammatory dry eye. Mechanism: T-cell modulation improves tear quality over weeks to months. PubMed

8. Lifitegrast 5% (twice daily)
   Purpose: Inflammatory dry eye. Mechanism: Blocks LFA-1/ICAM-1 interaction to reduce surface inflammation. PubMed

9. Antibiotics for bacterial dacryoadenitis (e.g., amoxicillin-clavulanate; dose per clinician and local resistance)
   Purpose: Treat proven infection. Mechanism: Kills bacteria. Note: Only if infection is diagnosed. NCBI

10. Short oral steroid taper for autoimmune dacryoadenitis/TED (specialist-guided)
    Purpose: Break significant inflammation that worsens bulge. Mechanism: Systemic immune suppression; requires careful screening and monitoring. endocrinologia.org.mx

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Regenerative / stem-cell–related” options

There are no approved stem-cell drugs that repair the position of a prolapsed lacrimal gland. The items below are used for associated diseases or ocular surface healing, not as a cure for prolapse.

1. Prednisone (systemic corticosteroid)
   Dose: Varies widely (e.g., 0.5–1 mg/kg/day short term) under specialist care. Function/Mechanism: Potent anti-inflammatory for autoimmune dacryoadenitis or active TED. Risks: Glucose rise, mood, infection. endocrinologia.org.mx

2. Mycophenolate (mofetil or sodium)
   Dose: Often 500–1000 mg twice daily (or sodium 0.72 g/day) as a steroid-sparing agent in TED. Function: T- and B-cell suppression. Evidence: Endorsed with IV steroids for moderate-to-severe active TED. endocrinologia.org.mx

3. Rituximab (anti-CD20)
   Dose: Protocols vary (e.g., 1000 mg IV ×2, 2 weeks apart). Function: Depletes B cells; used in refractory autoimmune orbitopathy/IgG4 disease that can involve the lacrimal gland. Note: Specialist decision only. endocrinologia.org.mx

4. Teprotumumab (IGF-1R inhibitor) for active TED
   Dose: IV infusions q3weeks ×8. Function: Reduces orbital inflammation and proptosis in TED. Relation to prolapse: Can improve the overall inflammatory orbit picture but does not “reposition” a prolapsed gland. Oxford Academic

5. Autologous serum tears (AST)
   Dose: Commonly 20% (varies), 4–8×/day. Function: Patient’s own growth factors aid ocular surface healing; helpful in severe dry eye or exposure symptoms. Status: Compounded; off-label.

6. Platelet-rich plasma (PRP) eye drops
   Dose: Protocols vary. Function: Delivers platelet-derived growth factors to support surface healing. Status: Off-label, growing use for severe ocular surface disease; not a prolapse fix.

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Surgeries

1. Lacrimal gland repositioning (dacryoadenopexy).
   Procedure: Through an upper-lid crease or inner-lid (transconjunctival) incision, the surgeon gently frees the gland and sutures it back to sturdy tissue (periosteum) near the outer orbital rim (often at Whitnall’s tubercle). Why: This restores normal position and smooths the lid contour while preserving tear function. WebEyeAmerican Academy of OphthalmologyPubMed

2. Upper blepharoplasty with gland preservation/reposition.
   Procedure: Removes redundant skin/fat and secures the prolapsed gland in one sitting. Why: Addresses both “excess skin” and the bulge safely. American Academy of Ophthalmology

3. Ptosis repair + gland reposition (when the outer lid margin is droopy).
   Procedure: Levator advancement plus dacryoadenopexy. Why: Corrects lid height and bulge together. Healio Journals

4. Lateral canthopexy/canthoplasty (select cases).
   Procedure: Tightens the outer lid corner if it’s lax. Why: Supports lid position and contour.

5. Partial dacryoadenectomy and biopsy (only when a mass is suspected).
   Procedure: Carefully removes a portion for diagnosis and to relieve mass effect. Why: Rules out tumor and treats confirmed benign lesions—not routine for simple prolapse. NCBI

Typical recovery: Swelling/bruising for 1–2 weeks; heavy exercise paused briefly; tear production is usually preserved if the gland is handled gently. Risks include recurrence (uncommon), dryness if the gland is damaged, scarring, or asymmetry. American Academy of Ophthalmology

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Prevention tips

1. Treat allergies early each season.

2. Avoid eye rubbing; use chilled tears instead.

3. Manage screen time; blink breaks.

4. Sleep slightly head-up.

5. Keep salt intake moderate, especially at night.

6. Stop smoking and avoid second-hand smoke. PMC

7. Use humidifiers in dry rooms.

8. Wear wraparound sunglasses on windy/sunny days.

9. If you ever have eyelid surgery, ask the surgeon to evaluate and secure any prolapsed gland during the same operation. American Academy of Ophthalmology

10. Keep thyroid disease well controlled if you have it. PMC

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When to see a doctor

• The bulge is new, rapidly growing, painful, or firm.

• There’s double vision, vision loss, or the eye looks pushed forward.

• You have fever or signs of infection.

• You have known thyroid disease with new eye changes.

• The bulge or asymmetry bothers you and you’re considering a fix. NCBI

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What to eat and what to avoid

Helpful to eat:

1. Plenty of water—dry indoor air + screens = thirsty eyes.

2. Fish rich in omega-3 (2–3 times/week): hilsa, sardine, mackerel—good for general inflammation (supplements show mixed benefit for dry eye). PubMedReview of Optometry

3. Leafy greens & colored veggies (vitamin A precursors for surface health).

4. Citrus and berries (vitamin C for collagen).

5. Nuts/legumes (vitamin E, plant proteins).

6. Foods with selenium (fish, eggs, whole grains)—especially relevant if you have mild active thyroid eye disease and your region/diet is low in selenium; your doctor may advise 100 µg twice daily for 6 months. New England Journal of MedicinePMC

Better to limit:
7) Excess salt at night (morning puffiness).
8) Alcohol excess (fluid shifts, sleep disruption).
9) Ultra-processed, high-sugar foods (systemic inflammation).
10) Smoking—harms healing and worsens TED. PMC

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FAQs

1) Can a prolapsed lacrimal gland turn into cancer?
No. Prolapse itself is not cancer. However, a new, firm, painful, or fast-growing mass needs urgent evaluation to rule out tumors that arise in or near the gland. NCBI

2) Will eye exercises or massage push it back?
No. Exercises don’t restore stretched supports, and pressing may irritate the gland.

3) Do eye drops cure prolapse?
No. Drops help comfort and the ocular surface but don’t change the gland’s position. PubMed

4) Is surgery always necessary?
Not always. If the bulge is small and you’re not bothered, observation is fine. Surgery is chosen for appearance, persistent irritation, or if tumor is suspected. American Academy of Ophthalmology

5) What surgery works best?
Repositioning (dacryoadenopexy) that preserves and secures the gland back to the periosteum is standard and aims to keep tear function normal. WebEyePubMed

6) What’s recovery like?
Bruising/swelling 1–2 weeks; most people resume normal activities soon after their surgeon’s clearance; tear function typically stays fine. American Academy of Ophthalmology

7) Can it come back after surgery?
Recurrence is uncommon but possible if tissues are very lax or if allergies/rubbing continue.

8) Will surgery make my eyes dry?
Good technique preserves the gland; dryness risk is low but discussed as a possible complication. American Academy of Ophthalmology

9) I have thyroid eye disease—can medicines help my bulge?
TED medicines (steroids, mycophenolate, teprotumumab) can calm inflammation but won’t “reposition” the gland; surgery fixes position. Selenium (100 µg twice daily ×6 months) helps mild active TED in low-selenium regions. endocrinologia.org.mxNew England Journal of Medicine

10) Is this the same as fat prolapse?
No. Fat prolapse comes from orbital fat herniating forward; imaging shows fat continuous with deeper fat. The lacrimal gland is a tear-making tissue, not fat. Review of OptometryPMC

11) Can kids get it?
Rarely—seen in certain congenital skull conditions or after trauma/surgery. Healio Journals

12) Could allergy shots help?
Treating allergies can lessen swelling and rubbing, which helps symptoms—but it won’t reverse prolapse.

13) Are omega-3 capsules worth it?
Evidence is mixed for dry eye; a large trial found fish oil wasn’t better than placebo. Eating fish is still healthy. PubMed

14) Will losing weight help?
It may reduce general puffiness and inflammation, which can make the bulge less noticeable, but it won’t reseat the gland.

15) Who should do the surgery?
An oculoplastic surgeon (an ophthalmologist with eyelid/orbit training).

Disclaimer: Each person’s journey is unique, treatment plan, life style, food habit, hormonal condition, immune system, chronic disease condition, geological location, weather and previous medical  history is also unique. So always seek the best advice from a qualified medical professional or health care provider before trying any treatments to ensure to find out the best plan for you. This guide is for general information and educational purposes only. Regular check-ups and awareness can help to manage and prevent complications associated with these diseases conditions. If you or someone are suffering from this disease condition bookmark this website or share with someone who might find it useful! Boost your knowledge and stay ahead in your health journey. We always try to ensure that the content is regularly updated to reflect the latest medical research and treatment options. Thank you for giving your valuable time to read the article.

The article is written by Team RxHarun and reviewed by the Rx Editorial Board Members

Last Updated: August 10, 2025.